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Scientists have been aware of a specific organelle in plant cells for more than a hundred years. However, UC Riverside scientists have only now discovered the organelle’s key role in aging.

The researchers initially set out to understand more generally which parts of plant cells control plant responses to stress from things like infections, too much salt, or too little light. Serendipitously, they found this organelle, and a protein responsible for maintaining the organelle, control whether plants survive being left too often in the dark.

Because they had not expected this discovery, which is described in a Nature Plants journal article, the research team was thrilled.

A genetic marker linked to premature aging was reversed in children with obesity during a six-month diet and exercise program, according to a recent study led by the Stanford School of Medicine.

Children’s telomeres—protective molecular “caps” on the chromosomes—were longer during the weight management program, then were shorter again in the year after the program ended, the study found. The research was published last month in Pediatric Obesity.

Like the solid segment at the end of a shoelace, telomeres protect the ends of chromosomes from fraying. In all people, telomeres gradually shorten with aging. Various conditions, including obesity, cause premature shortening of the telomeres.

Mainly this is about vertical farming.


In this eye-opening video, we explore the complex Environmental Impacts of an Aging Cure, delving into how extending Human Lifespan and pursuing Longevity could reshape our planet. We investigate the potential for increased Population Growth, the challenges of Sustainability, and the implications for Resource Consumption. Our analysis covers the Ecological Footprint of a world where aging is a thing of the past, addressing both the ethical dilemmas and the potential for Biomedical Advances in Age-Related Research. As concerns about Overpopulation and the need for Renewable Resources come to the forefront, we examine Eco-friendly Technologies and their role in supporting an age-extended society. Join us in this critical discussion about the intersection of Environmental Ethics and the quest for Age Extension.

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“If we give it to aged mice, they rejuvenate. If we give it to young mice, they age slower. No other therapy right now can do this.”


The fountain of youth has eluded explorers for ages. It turns out the magic anti-aging elixir might have been inside us all along.

Cold Spring Harbor Laboratory (CSHL) Assistant Professor Corina Amor Vegas and colleagues have discovered that T cells can be reprogrammed to fight aging, so to speak. Given the right set of genetic modifications, these white blood cells can attack another group of cells known as senescent cells. These cells are thought to be responsible for many of the diseases we grapple with later in life.

Senescent cells are those that stop replicating. As we age, they build up in our bodies, resulting in harmful inflammation. While several drugs currently exist that can eliminate these cells, many must be taken repeatedly over time.

Year 2021 Biocomputing is the future for the biological singularity because we could control all inputs and outputs of our bodies even evolve them eventually.


A silicon device that can change skin tissue into blood vessels and nerve cells has advanced from prototype to standardized fabrication, meaning it can now be made in a consistent, reproducible way. As reported in Nature Protocols, this work, developed by researchers at the Indiana University School of Medicine, takes the device one step closer to potential use as a treatment for people with a variety of health concerns.

The technology, called tissue nanotransfection, is a non-invasive nanochip device that can reprogram tissue function by applying a harmless electric spark to deliver specific genes in a fraction of a second. In laboratory studies, the device successfully converted into to repair a badly injured leg. The technology is currently being used to reprogram tissue for different kinds of therapies, such as repairing caused by stroke or preventing and reversing nerve damage caused by diabetes.

“This report on how to exactly produce these tissue nanotransfection chips will enable other researchers to participate in this new development in ,” said Chandan Sen, director of the Indiana Center for Regenerative Medicine and Engineering, associate vice president for research and Distinguished Professor at the IU School of Medicine.

The fountain of youth has eluded explorers for ages.


Summary: Researchers found that T cells can be genetically reprogrammed to target and eliminate senescent cells, which contribute to aging-related diseases. By using CAR (chimeric antigen receptor) T cells in mice, they achieved significant health improvements including lower body weight, enhanced metabolism, and increased physical activity.

This groundbreaking approach, offering long-term effects from a single treatment, could revolutionize treatments for age-related conditions like obesity and diabetes, transcending the potential of CAR T cells beyond their current use in cancer therapy.