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Luteolin, an antioxidant in vegetables, may contribute to the prevention of hair graying

Graying hair is a hallmark of aging, often considered an inevitable part of growing older. However, recent research from Nagoya University in Japan led by Masashi Kato and Takumi Kagawa suggests that an antioxidant might suppress this process. The researchers identified luteolin, an antioxidant found in vegetables including celery, broccoli, carrots, onions, and peppers, as being a potential anti-graying agent. Their findings pave the way for potential applications in human hair care.

The researchers’ study focused on three antioxidants—luteolin, hesperetin, and diosmetin—to assess their anti-graying effects in mice that were bred to go gray like humans. The difference was startling, the mice that received luteolin retained their black fur, even as their cage mates’ fur turned gray, regardless of whether the luteolin was given externally or internally.

“This result was surprising,” Professor Kato said. “While we expected that antioxidants may also have anti-graying effects, only luteolin, not hesperetin or diosmetin, demonstrated significant effects. This finding suggests that luteolin may have a unique medicinal effect that prevents graying.”

Vetek Associaiton — an organizer of international conferences in 2025

In 2025, Vetek Association is a founding partner in organizing several international conferences. In addition to the main longevity event in Israel – the Longevity Nation conference that will take place in Bar-Ilan University, on June 25–26 https://longevitynation.org/ Registration https://longevitynation.org/register-and-donate/

And community meetups in Israel https://www.longevityisrael.org/meetups/.

A mortality timer based on nucleolar size triggers nucleolar integrity loss and catastrophic genomic instability

Gutierrez and Tyler investigate the limits of replicative lifespan in yeast. The authors show that nucleolar expansion during aging is a mortality timer. Enlargement of nucleoli beyond a defined size alters their biophysical properties; normally excluded DNA repair protein enter, causing aberrant rDNA recombination, genome instability and death.

Telomeres Lengthened by 47% & Continue to Grow with Gene Therapy

Interesting item on biological clocks, and other things.


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Here Liz talks about her personal experience with gene therapy and the innovations, the state of the industry and what the future holds. The therapies exist why is it so difficult to make them available!?
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Akkermansia https://pendulumtherapeutics.sjv.io/b… Daily https://pendulumtherapeutics.sjv.io/N… OmegaQuant 5% discount Code MODERN https://omegaquant.com/shop/ Bulletproof 15% off with coupon code: HEALTHSPAN15: https://tinyurl.com/4npjk5vp Inner Fuel Gut support https://bulletproof.fdf2.net/PyDKDM Omega-3 Krill Oil https://bulletproof.fdf2.net/xkdxmy OneSkin 15% Discount: Code MODERN https://tinyurl.com/3t6tevj8 OS-01 Face Topical Supplement https://tinyurl.com/29c8wrr2 ⏲️Chapters 00:00 Liz’s interventions 12:40 Biological age tests and meaning 19:39 The paradigm of disease 23:30 Telomere results 26:00 Has she tried other therapies 30:20 Getting gene therapy now 40:50 Investment in longevity 47:10 CMV 50:40 Archie’s requirement for gene therapy 54:00 What is the future of BioViva & Gene therapy 1:01:00 Further information 🌐Links in this video BioViva Home Page https://bioviva-science.com/ BioViva on LinkedIn / biovivasciences Liz on X https://twitter.com/parrishliz BioViva on X https://twitter.com/BioVivaScience *************************************** Health claims Disclosure: Information provided on this video is not a substitute for direct, individual medical treatment or advice. Please consult with your doctor first. Products or services mentioned in this video are not a recommendation. Audio Copyright Disclaimer Please note that we have full authorization to the music that we used in our videos as they were created using the service WeVideo which provides the rights to the music. The rights are detailed in the terms of use that can be reviewed here https://www.wevideo.com/terms-of-use and any following inquiries should be addressed to [email protected]. ************************************************** #Lizparrish #genetherapy #telomeres.
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⏲️Chapters.
00:00 Liz’s interventions.
12:40 Biological age tests and meaning.
19:39 The paradigm of disease.
23:30 Telomere results.
26:00 Has she tried other therapies.
30:20 Getting gene therapy now.
40:50 Investment in longevity.
47:10 CMV
50:40 Archie’s requirement for gene therapy.
54:00 What is the future of BioViva & Gene therapy.
1:01:00 Further information.

🌐Links in this video.

A newly discovered mechanism rejuvenates aging cells

There are a multitude of products for sale that promise the appearance of eternal youth by erasing wrinkles or firming up jaw lines; but what if we could truly turn back time, at the cellular level? Now, researchers from Japan have found a protein that may do just that.

In a study published this month in Cellular Signaling, researchers from Osaka University have revealed that a key protein is responsible for toggling between ‘young’ and ‘old’ cell states.

As we age, older, fewer active cells, known as senescent cells, accumulate in multiple organs. These cells are noticeably larger than younger cells, and exhibit altered organization of stress fibers, the structural parts of cells that help them move and interact with their environment.

Key protein that toggles between ‘young’ and ‘old’ states may hold key to reversing cell aging

There are a multitude of products for sale that promise the appearance of eternal youth by erasing wrinkles or firming up jaw lines; but what if we could truly turn back time, at the cellular level? Now, researchers from Japan have found a protein that may do just that.

In a study published this month in Cellular Signaling, researchers from Osaka University have revealed that a key protein is responsible for toggling between “young” and “old” cell states.

As we age, older and less active cells, known as senescent cells, accumulate in multiple organs. These cells are noticeably larger than younger cells, and exhibit altered organization of fibers, the structural parts of cells that help them move and interact with their environment.

Viva City Fountainside Chat with Robin Hanson

Hey everyone! Robin Hanson will be speaking on Thursday about his galaxy brain ideas on better incentive models for longevity. Plus his unique takes on prediction markets and long-term thinking. [ https://lu.ma/wzuwk1lp](https://lu.ma/wzuwk1lp)


Join us for a groundbreaking discussion with economist Robin Hanson on the future of longevity economics and city governance!

Bioengineers reveal key to reversing cellular aging

“Our findings suggest that senescent cells maintain their large size through improved adhesion to the extracellular matrix via AP2A1 and integrin β1 movement along enlarged stress fibers,” Chantachotikul said.

The link between AP2A1 and senescent cells, the researchers said, means the protein has the potential to be used as a marker for cellular aging.

The team also believes that the findings may offer a new target for future treatments of age-related diseases.

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