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๐€ ๐๐ซ๐ฎ๐  ๐ญ๐ก๐š๐ญ ๐ข๐ง๐œ๐ซ๐ž๐š๐ฌ๐ž๐ฌ ๐๐จ๐ฉ๐š๐ฆ๐ข๐ง๐ž ๐œ๐š๐ง ๐ซ๐ž๐ฏ๐ž๐ซ๐ฌ๐ž ๐ญ๐ก๐ž ๐ž๐Ÿ๐Ÿ๐ž๐œ๐ญ๐ฌ ๐จ๐Ÿ ๐ข๐ง๐Ÿ๐ฅ๐š๐ฆ๐ฆ๐š๐ญ๐ข๐จ๐ง ๐จ๐ง ๐ญ๐ก๐ž ๐›๐ซ๐š๐ข๐ง ๐ข๐ง ๐๐ž๐ฉ๐ซ๐ž๐ฌ๐ฌ๐ข๐จ๐ง, ๐„๐ฆ๐จ๐ซ๐ฒ ๐ฌ๐ญ๐ฎ๐๐ฒ ๐ฌ๐ก๐จ๐ฐ๐ฌ

๐˜ผ๐™ฃ ๐™€๐™ข๐™ค๐™ง๐™ฎ ๐™๐™ฃ๐™ž๐™ซ๐™š๐™ง๐™จ๐™ž๐™ฉ๐™ฎ ๐™จ๐™ฉ๐™ช๐™™๐™ฎ ๐™ฅ๐™ช๐™—๐™ก๐™ž๐™จ๐™๐™š๐™™ ๐™ž๐™ฃ ๐™‰๐™–๐™ฉ๐™ช๐™ง๐™šโ€™๐™จ ๐™ˆ๐™ค๐™ก๐™š๐™˜๐™ช๐™ก๐™–๐™ง ๐™‹๐™จ๐™ฎ๐™˜๐™๐™ž๐™–๐™ฉ๐™ง๐™ฎ ๐™จ๐™๐™ค๐™ฌ๐™จ ๐™ก๐™š๐™ซ๐™ค๐™™๐™ค๐™ฅ๐™–, ๐™– ๐™™๐™ง๐™ช๐™œ ๐™ฉ๐™๐™–๐™ฉ ๐™ž๐™ฃ๐™˜๐™ง๐™š๐™–๐™จ๐™š๐™จ ๐™™๐™ค๐™ฅ๐™–๐™ข๐™ž๐™ฃ๐™š ๐™ž๐™ฃ ๐™ฉ๐™๐™š ๐™—๐™ง๐™–๐™ž๐™ฃ, ๐™๐™–๐™จ ๐™ฅ๐™ค๐™ฉ๐™š๐™ฃ๐™ฉ๐™ž๐™–๐™ก ๐™ฉ๐™ค ๐™ง๐™š๐™ซ๐™š๐™ง๐™จ๐™š ๐™ฉ๐™๐™š ๐™š๐™›๐™›๐™š๐™˜๐™ฉ๐™จ ๐™ค๐™› ๐™ž๐™ฃ๐™›๐™ก๐™–๐™ข๐™ข๐™–๐™ฉ๐™ž๐™ค๐™ฃ ๐™ค๐™ฃ ๐™—๐™ง๐™–๐™ž๐™ฃ ๐™ง๐™š๐™ฌ๐™–๐™ง๐™™ ๐™˜๐™ž๐™ง๐™˜๐™ช๐™ž๐™ฉ๐™ง๐™ฎ, ๐™ช๐™ก๐™ฉ๐™ž๐™ข๐™–๐™ฉ๐™š๐™ก๐™ฎ ๐™ž๐™ข๐™ฅ๐™ง๐™ค๐™ซ๐™ž๐™ฃ๐™œ ๐™จ๐™ฎ๐™ข๐™ฅ๐™ฉ๐™ค๐™ฃ๐™จ ๐™ค๐™› ๐™™๐™š๐™ฅ๐™ง๐™š๐™จ๐™จ๐™ž๐™ค๐™ฃ.

Numerous labs across the world have shown that inflammation causes reduced motivation and anhedonia, a core symptom of depression, by affecting the brainโ€™s reward pathways.


An Emory University study published in Natureโ€™s Molecular Psychiatry shows levodopa, a drug that increases dopamine in the brain, has potential to reverse the effects of inflammation on brain reward circuitry, ultimately improving symptons of depression.

Past research conducted by the Department of Psychiatry and Behavioral Sciences at Emory University School of Medicine has linked the effects of inflammation on the brain to decreased release of dopamine, a chemical neurotransmitter that regulates motivation and motor activity, in the ventral striatum.